General Fitness

Exercise Through the Ages

The 82-Year-Old Who Quietly Changed the Conversation

Earlier this year, The Washington Post profiled Juan López García, an 82-year-old ultramarathon runner from Toledo whose physiology has caught the attention of researchers across Europe.

His VO₂ max sits at a level typically seen in healthy adults decades younger. His aerobic capacity is the highest ever recorded in someone in their 80s. What makes the story more compelling is that he did not grow up as an elite endurance athlete. He began running seriously in his mid-60s after retiring from his job as a mechanic.

 

The headline is not that he is extraordinary. The headline is that adaptation continued.

The Washington Post article highlighted lab testing carried out by Spanish and Italian researchers, showing that his cardiovascular system, muscle oxygen extraction and endurance capacity remain highly developed well into his eighth decade. That profile challenges the assumption that ageing inevitably shuts down meaningful improvement.

It does not.

And the wider research over the past two years supports that point strongly.

Building Capacity in Early Adulthood

In our 20s and 30s, most physiological systems are near their natural peak. VO₂ max, muscle mass, bone density and neuromuscular efficiency are typically at their highest levels. This stage of life offers a powerful opportunity to build physiological reserve.

Longitudinal tracking studies from Scandinavian cohorts have followed participants for more than four decades. Data published by researchers at the Karolinska Institutet in 2026 showed that physical performance begins a gradual decline in the mid-30s, yet individuals who maintained consistent physical activity retained significantly higher aerobic capacity and muscular strength across the lifespan. Active participants showed a slower rate of decline and better preserved functional performance in later life.

Exercise in early adulthood builds:

  • Greater mitochondrial density
  • Higher stroke volume and cardiac output
  • Increased capillarisation
  • Stronger bones through mechanical loading
  • Improved insulin sensitivity

These adaptations form a reserve. That reserve has lasting effects, even decades later.

Bone mineral density research consistently shows that peak bone mass achieved in early adulthood predicts fracture risk in older age (Weaver et al., 2016). Resistance training during these decades influences long-term musculoskeletal health in a measurable way.

The foundation phase is not about aesthetics. It is about building capacity that remains biologically relevant long after competitive sport has ended.

Slowing the Curve in Midlife

It is unfortunate that the narrative around midlife often becomes unnecessarily fatalistic. Decline is discussed as though it happens in large, fixed steps.

But that’s not true. Research paints a totally different picture.

Aerobic capacity typically declines by around 10% per decade after the age of 30 in sedentary adults (Fleg et al., 2005). Longitudinal endurance athlete data show much smaller reductions when training volume is maintained. The difference between active and inactive adults at 60 years old can exceed 20 ml/kg/min in VO₂ max. That gap represents a substantial difference in mortality risk, given that cardiorespiratory fitness is one of the strongest predictors of all-cause mortality (Blair et al., 1989; Kodama et al., 2009).

Brain health research has strengthened this argument in recent years. A 2025 study published in JAMA Network Open by researchers at Boston University found that higher levels of moderate-to-vigorous physical activity in mid- or late life were associated with approximately 41–45% lower dementia risk (Morino et al., 2025). Importantly, participants who increased activity levels later in life also saw reduced risk.

Johns Hopkins researchers reported similar findings in 2025, showing that even modest amounts of daily moderate-to-vigorous activity were associated with significant reductions in dementia risk compared with sedentary behaviour (Wanigatunga et al., 2025).

At the muscular level, resistance training remains effective. Age-related sarcopenia begins in midlife, yet systematic reviews continue to show meaningful hypertrophy and strength gains in adults well into their 60s and 70s (Peterson et al., 2010; Tieland et al., 2018). Muscle protein synthesis remains responsive to mechanical tension.

Midlife is not a closing door. It is a stage where training decisions have long-term health implications.

Adaptation Continues in Later Life

The assumption that older adults can only maintain is also not supported by current research and evidence.

Systematic reviews published in 2026 examining structured exercise interventions in adults over 70 demonstrate consistent improvements in strength, gait speed, balance and functional capacity following 12 weeks or more of progressive training. Improvements in muscle strength frequently exceed 20–30% in previously untrained older adults (Liu & Latham, 2009; updated analyses 2026).

Resistance training increases type II fibre recruitment even in advanced age. Mitochondrial function improves with endurance training. Insulin sensitivity responds to activity at 75 in much the same mechanistic way it does at 35.

Juan López García represents an extreme case of preserved aerobic capacity. His physiology may include favourable genetics. The key point remains that training stimulated adaptation well after retirement age.

His laboratory data showed high muscle oxygen extraction capacity and efficient energy utilisation. That profile reflects long-term training stimulus.

Ageing does not remove plasticity. It reduces the margin for inactivity.

The Brain, Inflammation and Biological Ageing

As we all know, exercise influences systems beyond muscle and heart.

A 2025 review in the Journal of Sport and Health Science described exercise as a modulator of multiple hallmarks of ageing, including mitochondrial dysfunction, chronic low-grade inflammation and impaired autophagy (Qui et al., 2025). Regular physical activity improves mitochondrial biogenesis via PGC-1α signalling. It reduces inflammatory cytokine levels. It enhances endothelial function.

Exercise variety also appears relevant. A study published in the 2026 BMJ Journals found that greater diversity in exercise types was associated with a 19% lower risk of premature mortality, independent of total volume (Han et al., 2026).

The biological message is clear. Movement influences systemic health markers tied directly to longevity.

Functional Independence and Quality of Life

Beyond laboratory markers, physical independence is central to healthy ageing.

Multicomponent training programmes combining resistance work, balance drills and aerobic exercise significantly reduce fall risk and improve activities of daily living in older adults (Sherrington et al., 2019; updated meta-analysis 2026).

Gait speed alone predicts survival in older populations (Studenski et al., 2011). Structured exercise improves gait speed.

These improvements are not trivial. They represent the ability to live independently.

For professionals working in the field, understanding these adaptations is essential knowledge. Today’s personal trainer courses increasingly emphasise lifespan physiology for that reason. Programming knowledge across decades is more important than ever for PTs. It informs safe progression and realistic goal setting.

Even in a strength & conditioning course, discussions around long-term athlete development now include masters populations and late-life adaptation models. The science supports inclusion across age brackets.

The fundamentals taught in a basic pt course around progressive overload, recovery management and individualisation apply at 70 just as they do at 30. The variables shift. The principles remain.

The “Too Late” Myth

As a coach, you may have heard the phrase “I should have started earlier” before.

Earlier exposure builds a larger reserve. That is true.

Later starts however, still produce measurable physiological improvement. This is equally true.

Muscle fibres respond to tension at 75. The cardiovascular system responds to endurance training at 80. The brain responds to increased blood flow and neurotrophic signalling in later life.

The rate of decline can be slowed.

The human organism remains adaptable for far longer than cultural narratives suggest, as seen in our ultra marathon runner, Juan López García.

Practical Implications

The research points towards several consistent themes:

  • Aerobic capacity remains a powerful predictor of health outcomes at every age.
  • Resistance training protects muscle mass, metabolic function and independence.
  • Even modest increases in activity reduce dementia risk.
  • Training variety may support broader longevity benefits.
  • Consistency across decades produces compounding returns.

There is no age threshold beyond which exercise loses relevance.

Final Thoughts

Juan López García did not eliminate ageing. He demonstrated that, regardless of age, adaption still occurs.

Longitudinal cohort data, dementia research, systematic reviews on resistance training and biological ageing analyses all converge on the same message: exercise delivers benefits across the lifespan.

At 25, the body adapts.
At 55, the body adapts.
At 85, the body adapts.

The stimulus still works.

And the evidence base is indisputable.

So next time a client tells you they’re too old to start, you’ll have the research and the confidence to show them that age is not the barrier they think it is.

Reference

Train to Coach Every Age with Confidence

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